Subido por Medicina Icesi 16-2

3. P

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4. LA ONDA P Y SUS ALTERACIONES.
ARRITMIAS SUPRAVENTRICULARES
FELIPE CAÑAS ORDUZ
FELLOW CARDIOLOGIA
ICESI
2018
Figure 2: Schematic of normal atrial activation. Atrial activation starts at the sinus
node near the right atrial-superior vena cava (SVC) junction. The right atrium is
activated first, which is then followed by left atrial activation. For this reason, in
general the initial part of the P wave reflects right atrial activation and the terminal
portion of the P wave is due to left atrial activation (reprinted with permission from
Kusumoto FM, Cardiovascular Pathophysiology, Hayes Barton Press, Raleigh, NC,
1999).
CRECIMIENTO AURICULAR
1. Anatomía cardíaca, registro
electrocardiográfico y análisis de eje.
however, indicates that the finding of a tall, peaked
P wave does not consistently correlate with RAA.
Fig. 7.1 The normal P wave is usually less than 2.5 mm in
pulmo
acquire
opathy
cardiom
nary di
pu lm o
bronch
hypert
lung di
nia). C
in clud
defects
tricusp
also be
includi
stenosi
carcino
height and less than 0.12 sec in width.
Right Atrial Abnormality (Overload)
LEFT
ABNO
Enlarge
ONDA P NORMAL
Duración
80 – 120 ms
Eje
0 - 75°
Positiva
DI, DII, AVF
Bifásica
DIII, AVL, V1, V2
Amplitud
< 2.5mm.VI: (+) <1.5mm (-) < 1mm
P PULMONAR
ht atrium (RA) may cause
tremity or chest leads. An
LA) may cause broad, often
mity leads and a biphasic P
nent negative component
ization of the left atrium.
ntheroth WG: How to read
uis, Mosby/ Elsevier, 2006.)
Atrial Enlargement (Abnormality)
LA
RA
V1
Normal
Right
Left
RA
II
RA
LA
LA
RA
RA
LA
RA
RA
V1
LA
LA
LA
ARRITMIAS SUPRAVENTRICULARES
ARRITMIAS SUPRAVENTRICULARES
ARRITMIAS SUPRAVENTRICULARES
1. Arritmias sinusales
2. Arritmia atriales
3. Nodo sinusal enfermo
4. Ritmos de escape
5. Reentrada intranodal
6. Preexitación
7. Fibrilación auricular
8. Flutter atrial
1. ARRITMIAS SINUSALES
BRADICARDIA SINUSAL
CAUSAS DE BRADICARDIA SINUSAL
Fisiológica
SAHOS
Medicamentos
Infarto
Hiperkalemia
Endocrinopatías
Epilepsia
Nodo sinusal
enfermo
TAQUICARDIA SINUSAL
CAUSAS DE TAQUICARDIA SINUSAL
Fisiológica
Dolor
Medicamentos
Alcohol
Fiebre
Hipovolemia
Falla cardíaca
TEP
Infarto
Epilepsia
Endocrinopatías
ARRITMIA RESPIRATORIA
SINUSAL
2. ARRITMIAS ATRIALES
RITMO ATRIAL BAJO
SENO
CORONARIO
EXTRASISTOLIA ATRIAL
3. NODO SINUSAL ENFERMO
Table 9.1 Etiologies of sinus node dysfunction.
Categories
Specific etiologies
Intrinsic
Rheumatologic diseases
Rheumatic fever, scleroderma, ankylosing
spondylitis, Reiter’s syndrome, tuberous
sclerosis
Congenital diseases
Correction of congenital heart defects,
autosomal dominant sinus node dysfunction
Tumors
Lymphoma, granular cell tumor
SA nodal ischemia
Myocardial infarction, embolism
Infections
Chagas’ disease
Trauma
After cardiac surgery, penetrating cardiac
trauma
Infiltrative diseases
Sarcoidosis, amyloid, radiation therapy
Extrinsic
Autonomic responses
Normal response, exaggerated vagal tone
(carotid sinus hypersensitivity)
Electrolytes, hypoxia, and
hormones
Thyroid disease, hyperkalemia, hypothermia,
anorexia nervosa, hypoxia, sleep apnea
Medications
b-blockers, calcium channel blockers,
antiarrhythmics, chemotherapy, lithium,
phenothiazines, cimetidine, tricyclic
antidepressants
In fact, a number of investigators have demonstrated that the sinus node artery
4. RITMOS DE ESCAPE
UNION
5. REENTRADA INTRANODAL
?
6. PRE EXITACIÓN (WPW)
WPW en
sinusal
3q
WPW en
taquicardia:
Ortodrómica
QRS estrecho
WPW en
taquicardia:
Antidrómica
QRS ancho
7 FIBRILACION AURICULAR
atrial activation leads to irregular ventricular activation.
Figure 8: Cartoon showing one of the potential mechanisms of atrial fibrillation.
A rotor is a very localized reentrant circuit that appears to propagate “arms” of
activation. When the “arms” meet obstacles—a region of refractory tissue, a “hole”
such as a valve or vein opening—the arms split into “daughter” rotors.
The atrial rate is very rapid, 300 to 1000 beats per minute. Fortunately, the AV
node limits the ventricular response rate, although in younger patients fairly rapid
8. FLUTTER
FLUTTER ATRIAL
ARRITMIAS AURICULARES
RESUMEN
FORMACION DEL IMPULSO
Sinusal
Atrial
Union
CONDUCCION DEL IMPULSO
Flutter
Reentrada
nodal
Fibrilación
auricular
Pre exitación
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